Where do you find transcription factors?
Transcription factors function in the nucleus, where genes are found, and nuclear transport (i.e., import or export) of transcription factors can influence their activity. Another important general mechanism controlling the activity of transcription factors is posttranslational modification such as phosphorylation.
What is K protein?
Heterogeneous nuclear ribonucleoprotein K (also protein K) is a protein that in humans is encoded by the HNRNPK gene. It is found in the cell nucleus that binds to pre-messenger RNA (mRNA) as a component of heterogeneous ribonucleoprotein particles. The simian homolog is known as protein H16.
What are the ingredients in Special K?
Rice, wheat gluten, sugar, defatted wheat germ, contains 2% or less of salt, malt flavor, BHT for freshness.
Is Special K fortified?
Is Special K fortified with vitamins and minerals? Our Special K flake is fortified with vitamin D, a range of B vitamins (B1, B2, B3, B6, B9 & B12), iron and zinc. And, our core Special K Bars are fortified with vitamins B3, B6 & B9.
What is the main function of transcription factors?
Transcription factors (TFs) are proteins that bind specific regulatory DNA sequences, and regulate (activate or repress) the transcription of genetic information from DNA to messenger RNA (mRNA).
What is HNRNPK s379?
hnRNP K Is a Novel Internal Ribosomal Entry Site-Transacting Factor That Negatively Regulates Foot-and-Mouth Disease Virus Translation and Replication and Is Antagonized by Viral 3C Protease. hnRNPK S379 phosphorylation participates in migration regulation of triple negative MDA-MB-231 cells.
Is hnRNP K a transcriptional amplifier of D site-binding proteins?
RNA-binding motifs of hnRNP K are critical for induction of antibody diversification by activation-induced cytidine deaminase. Taken together, the results show rhythmic protein expression of hnRNP K and provide new insights into its function as a transcriptional amplifier of D site-binding protein.
Does HNRNPK mediate the ZRS variant regulation of Shh transcriptional activity?
Results provide evidence that HnRNPK mediates the ZRS variant regulating the transcriptional activity of SHH. As a novel regulator, lnc-LBCS plays an important tumor-suppressor role in BCSCs’ self-renewal and chemoresistance, contributing to weak tumorigenesis and enhanced chemosensitivity.
What is the relationship between HNRNPK and lncRNAs?
The hnRNPK-lncRNAs interaction is potentially implicated in various pathogenic disorders including tumorigenesis, and Kabuki-like, Au-Kline, and Okamoto syndromes. Role of heterogeneous nuclear ribonucleoprotein K in tumor development.