What are signs of post-cardiac arrest syndrome?

What are signs of post-cardiac arrest syndrome?

Syndrome Pathophysiology Clinical Manifestation
Post–cardiac arrest brain injury • Impaired cerebrovascular autoregulation • Seizures
• Cerebral edema (limited) • Myoclonus
• Postischemic neurodegeneration • Cognitive dysfunction
• Persistent vegetative state

Can hypoxia cause myoclonus?

Myoclonus may occur after hypoxia. In 1963, Lance and Adams described persistent myoclonus with other features after hypoxia. However, myoclonus occurring immediately after hypoxia may demonstrate different syndromic features from classic Lance–Adams syndrome (LAS).

Is post anoxic myoclonus a seizure?

Post-anoxic myoclonus is a movement disorder occurring after hypoxic brain events. Myoclonus in acute PAM is sometimes considered as an ictal phase of epileptic seizure. It is crucial to monitor patients with EEG in order to detect of epileptiform discharges before and after treatment.

What is Posthypoxic myoclonus?

Post-hypoxic myoclonus (PHM) refers to myoclonus occurring after hypoxic brain injury resulting from a cardiac arrest, characterised by abrupt, irregular contractions of muscles that may be focal or generalised.

What are the three signs of cardiac arrest?

Signs of sudden cardiac arrest are immediate and drastic and include: Sudden collapse. No pulse. No breathing….When to see a doctor

  • Chest pain or discomfort.
  • Heart palpitations.
  • Rapid or irregular heartbeats.
  • Unexplained wheezing.
  • Shortness of breath.
  • Fainting or near fainting.
  • Lightheadedness or dizziness.

How do you evaluate myoclonus?

To determine the cause of myoclonus and rule out other potential causes of your condition, your doctor may recommend several tests, including:

  1. Electroencephalography (EEG)
  2. Electromyography (EMG)
  3. Evoked potential studies.
  4. Magnetic resonance imaging (MRI)
  5. Laboratory tests.
  6. Medications.
  7. Therapies.
  8. Surgery.

What is cortical myoclonus?

Cortical myoclonus is defined as muscle jerks originating from abnormal electrical activity in the cerebral cortex, which can be observed on conventional EEG or demonstrated by EEG backaveraging [1], [2]. Cortical myoclonus may occur spontaneously, in response to somaesthetic stimuli or during movement.

What is action myoclonus?

Action myoclonus, reviewed in this chapter, is the term applied to arrhythmic muscular jerking induced by voluntary movement. It is made worse by attempts at precise or coordinated movement (intention myoclonus) and may also be provoked by certain sensory stimuli.

Does post cardiac arrest myoclonus have a universally poor prognosis?

In conclusion, it is important to recognise that post cardiac arrest myoclonus does not have a universally poor prognosis. It is vital to distinguish between myoclonic status epilepticus and the Lance–Adams syndrome. Table 1 Distinguishing features of myoclonic status epilepticus and Lance–Adams syndrome.

What is post-hypoxic myoclonus?

OVERVIEW Post-hypoxic myoclonus (PHM) refers to myoclonus occurring after hypoxic brain injury resulting from a cardiac arrest, characterised by abrupt, irregular contractions of muscles that may be focal or generalised

What is pediatric myoclonus and what causes it?

Pediatric myoclonus is a quick, involuntary, muscle jerk that starts and stops very quickly. The movement can be repetitive, but usually occurs in different places on the body in the same person. What is Pediatric Myoclonus?

What is pediatric post–cardiac arrest care?

Pediatric post–cardiac arrest care focuses on anticipating, identifying, and treating this complex physiology to improve survival and neurological outcomes.